Selected Publications

Functional analysis of a triplet deletion in the gene encoding the sodium glucose transporter 3, a potential risk factor for ADHD.
N. Schäfer, M. Friedrich, M.E. Jørgensen, S. Kollert, H. Koepsell, E. Wischmeyer, K.P. Lesch, D. Geiger, F. Döring (2018).
PlosOne 13(10):e0205109. doi: 10.1371.
Characterization of small fiber pathology in a mouse model of Fabry disease.
L. Hofmann, D. Hose, A. Grießhammer, R. Blum, F. Döring, S. Dib-Hajj, S. Waxman, C. Sommer, E. Wischmeyer, N. Üçeyler (2018).
eLife 7. pii: e39300. doi: 10.7554.
Nimodipine fosters remyelination in a mouse model of multiple sclerosis and induces microglia-specific apoptosis.
A. Schampel, O.Volovitch, T. Koeniger, C. Scholz, S. Jörg, R. Linker, E. Wischmeyer, M. Wunsch, J. Hell, S. Ergün, S. Kuerten (2017).
Proc. Natl. Acad. Sci. USA. 2017 Apr 18, E3295-E3304
Disruption of G-protein signaling by PTH receptor mutants isolated from patients with primary failure of tooth eruption.
H. Subramanian, S. Kollert, N. Rukoyatkina, J. Sturm, S. Gambaryan, A. Stellzig-Eisenhauer, P. Meyer-Marcotty, F. Döring, M. Eigenthaler, E. Wischmeyer (2016).
PlosOne (11), e0167033
Mitragynine and its potential blocking effects on specific cardiac potassium channels.
Y.L. Tay, Y.F. Teah, Y.M Chong, M.F. Jamil, S. Kollert, M.I. Adenan, H.A. Wahab, F. Döring, E. Wischmeyer, M.L. Tan (2016).
Toxicol. Appl. Pharmacol. (15), 22-39
Activation of TRESK channels by the inflammatory mediator lysophosphatidic acid balances nociceptive signalling.
S. Kollert, B. Dombert, F. Döring, E. Wischmeyer (2015).
Sci. Rep. 2015 (5),12548
Distribution and function of sodium channel subtypes in human atrial myocardium.
S.G. Kaufmann, R.E. Wesatenbroek, A.H. Maass, V. Lange, A. Renner, E. Wischmeyer, A. Bonz, J. Muck, G. Ertel, W.A. Catterall, T. Scheuer, S.K. Maier (2013).
J. Mol. Cell. Cardiol. (61),133-4
The connection of monocytes and reactive oxygen species in pain.
D. Hackel, D. Pflücke, A. Neumann, J. Viebahn, S. Mousa, E. Wischmeyer, N. Roewer, A. Brack, H. Rittner. (2013).
PLoS One, e6356
Ectopic expression of neurogenin 2 alone is sufficient to induce differentiation of embryonic stem cells into mature neurons.
E.C. Thoma, E. Wischmeyer, N. Offen, K. Maurus, A.L. Siren, M. Schartl, T.U. Wagner (2012).
PLoS One (8), e386
Heterodimerization of serotonin receptors 5-HT1A and 5-HT7 differentially regulates receptor signalling and trafficking.
U. Renner, A. Zeug, A. Woehler, M. Niebert, A. Dityatev, G. Dityateva, N Gorinski, D. Guseva, D. Abdel-Galil, M. Fröhlich, F. Döring, E. Wischmeyer, D.W. Richter , E. Neher, E.G. Ponimaskin (2012).
J. Cell. Sci. (125), 2486-99
TREK-1 isoforms generated by alternative translation initiation display different susceptibility to the antidepressant fluoxetine.
M. Eckert, B. Egenberger, F. Döring, E. Wischmeyer (2011).
Neuroparmacology (61), 918-923
Functional consequences of interactions between the neuronal cell adhesion molecule NCAM, the receptor tyrosine kinase TrkB and the inwardly rectifying K+ channel Kir3.3
R. Kleene, C. Cassens, R. Bähring, T. Theis, M.Xiang, J. Xu, A. Dityatev, C. Schafer-Nielsen, F. Döring, E. Wischmeyer, M. Schachner (2010).
J. Biol. Chem. 285 (37), 28968-28979
N-linked glycosylation determines cell surface expression of two-pore-domain K+ channel TRESK.
B. Egenberger, G. Polleichtner, E. Wischmeyer , F. Döring (2010).
Biochem. Biophys. Res. Commun. 391(2):1262-1267
Intracellular traffic of the K(+) channels TASK-1 and TASK-3: role of N- and C-terminal sorting signals and interaction with 14-3-3 proteins.
M. Zuzarte, K. Heusser, V. Reningunta, G. Schlichthörl, S. Rinné, E. Wischmeyer, J. Daut, B. Schwappach, R. Preisig-Müller (2009).
J. Physiol. 587 (5), 929-952
Excitability of pontine startle processing neurones is regulated by the two-pore domain K+ channels TASK-3 coupled to 5-HT2C receptors.
M. Weber, A. Schmitt, E. Wischmeyer, F. Döring (2008).
Europ. J. Neurosci. (28), 931-940
TRESK two-pore-domain K channels constitute a significant component of background potassium currents in murine dorsal root ganglion neurones.
T. Dobler, A. Springauf, S. Tovornik, M. Weber, A. Schmitt, R. Sedlmeier, E. Wischmeyer, F. Döring (2007).
J. Physiol. 585 (3), 867-879
Novel Drosophila two-pore domain K+ channels: rescue of channel function by heteromeric assembly.
F. Döring, H. Scholz, R. Kühnlein, A. Karschin and E. Wischmeyer (2006).
Europ. J. Neurosci. (24), 2264-2274
Inwardly rectifying K+ (Kir) channels in Drosophila.
F.Döring, E. Wischmeyer, R.P. Kühnlein, H. Jäckle, A. Karschin (2002).
J. Biol. Chem. 277(28), 25554-2556
The neural cell adhesion molecule NCAM regulates cell surface delivery of G-protein activated inwardly rectifying potassium channels via lipid rafts.
M. Delling, E. Wischmeyer, A. Dityatev, R. Veh, A. Karschin, M. Schachner (2002).
J. Neurosci. 22(16), 7154-7164
Acute suppression of inwardly rectifying Kir2.1 channels by direct tyrosine kinase phosphorylation.
E. Wischmeyer, F. Döring, A. Karschin (1998).
J. Biol. Chem. 273 (51),34063-34068
The epithelial inward rectifier Kir7.1 displays unusual K+ permeation properties.
F. Döring, C. Derst, E. Wischmeyer, C. Karschin, R. Schneggenburger, J. Daut , A. Karschin (1998).
J. Neurosci. 18 (21), 8625-8636
Kir2.4: a novel brain K+ inward rectifier predominantly expressed in motor neurons of cranial nerve nuclei.
C. Töpert , F. Döring, E. Wischmeyer, C. Karschin, J. Brockhaus, K. Ballanyi, C. Derst, A. Karschin (1998).
J. Neurosci. 18 (11), 4096-4105
Subunit interactions in the assembly of neuronal Kir 3.0 inwardly rectifying K+ channels.
E. Wischmeyer, F. Döring, E. Wischmeyer, A. Spauschus, A. Karschin (1997).
Mol. Cell. Neurosci. 8, 1-14
Receptor stimulation causes slow inhibition of IRK1 inwardly rectifying K+ channels by direct protein kinase A-mediated phosphorylation.
E. Wischmeyer, A. Karschin (1996).
P. Natl. Acad. Sci. USA 93, 5819-5823